Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice

Objective of the Study: Diabetic patients have a much more widespread and aggressive form of atherosclerosis and therefore, higher risk for myocardial infarction, peripheral vascular disease and stroke, but the molecular mechanisms leading to accelerated damage are still unclear. Recently, we showed...

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Main Author: Zetterqvist, Anna V. (author)
Other Authors: Berglund, Lisa M. (author), Blanco, Fabiana (author), Garcia-Vaz, Eliana (author), Wigren, Maria (author), Duner, Pontus (author), Dutius Andersson, Anna-Maria (author), Nilsson, Jan (author), Bengtsson, Eva (author), Spegel, Peter (author)
Format: article
Language:English
Published: 2013
Online Access:https://hdl.handle.net/20.500.12008/21978
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author Zetterqvist, Anna V.
author2 Berglund, Lisa M.
Blanco, Fabiana
Garcia-Vaz, Eliana
Wigren, Maria
Duner, Pontus
Dutius Andersson, Anna-Maria
Nilsson, Jan
Bengtsson, Eva
Spegel, Peter
author2_role author
author
author
author
author
author
author
author
author
author_browse Bengtsson, Eva
Berglund, Lisa M.
Blanco, Fabiana
Duner, Pontus
Dutius Andersson, Anna-Maria
Garcia-Vaz, Eliana
Nilsson, Jan
Spegel, Peter
Wigren, Maria
Zetterqvist, Anna V.
author_facet Zetterqvist, Anna V.
Berglund, Lisa M.
Blanco, Fabiana
Garcia-Vaz, Eliana
Wigren, Maria
Duner, Pontus
Dutius Andersson, Anna-Maria
Nilsson, Jan
Bengtsson, Eva
Spegel, Peter
author_role author
collection COLIBRI
dc.contributor.none.fl_str_mv Zetterqvist Anna V.
Berglund Lisa M.
Blanco Fabiana
Garcia-Vaz Eliana
Wigren Maria
Duner Pontus
Dutius Andersson Anna-Maria
Nilsson Jan
Bengtsson Eva
Spegel Peter
dc.creator.none.fl_str_mv Zetterqvist, Anna V.
Berglund, Lisa M.
Blanco, Fabiana
Garcia-Vaz, Eliana
Wigren, Maria
Duner, Pontus
Dutius Andersson, Anna-Maria
Nilsson, Jan
Bengtsson, Eva
Spegel, Peter
dc.date.none.fl_str_mv 2013
2019-09-27T17:50:07Z
2019-09-27T17:50:07Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv Zetterqvist, A, Berglund, L, Blanco, F, y otros. "Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice". PLoS ONE Vol.8, no.6 [en línea] 2013, doi:10.1371/journal.pone.0065020
https://hdl.handle.net/20.500.12008/21978
dc.language.none.fl_str_mv en
eng
dc.publisher.none.fl_str_mv PLOS ONE Editorial Board
dc.relation.none.fl_str_mv PLoS ONE Vol.8, no.6, 2013
dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
Licencia Creative Commons Atribución (CC - BY)
dc.source.none.fl_str_mv reponame:COLIBRI
instname:Universidad de la República
instacron:Universidad de la República
dc.title.none.fl_str_mv Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice
dc.type.none.fl_str_mv Artículo
info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
description Objective of the Study: Diabetic patients have a much more widespread and aggressive form of atherosclerosis and therefore, higher risk for myocardial infarction, peripheral vascular disease and stroke, but the molecular mechanisms leading to accelerated damage are still unclear. Recently, we showed that hyperglycemia activates the transcription factor NFAT in the arterial wall, inducing the expression of the pro-atherosclerotic protein osteopontin. Here we investigate whether NFAT activation may be a link between diabetes and atherogenesis. Methodology and Principal Findings: Streptozotocin (STZ)-induced diabetes in apolipoprotein E2/2 mice resulted in 2.2 fold increased aortic atherosclerosis and enhanced pro-inflammatory burden, as evidenced by elevated blood monocytes, endothelial activation- and inflammatory markers in aorta, and pro-inflammatory cytokines in plasma. In vivo treatment with the NFAT blocker A-285222 for 4 weeks completely inhibited the diabetes-induced aggravation of atherosclerosis, having no effect in non-diabetic mice. STZ-treated mice exhibited hyperglycemia and higher plasma cholesterol and triglycerides, but these were unaffected by A-285222. NFAT-dependent transcriptional activity was examined in aorta, spleen, thymus, brain, heart, liver and kidney, but only augmented in the aorta of diabetic mice. A- 285222 completely blocked this diabetes-driven NFAT activation, but had no impact on the other organs or on splenocyte proliferation or cytokine secretion, ruling out systemic immunosuppression as the mechanism behind reduced atherosclerosis. Instead, NFAT inhibition effectively reduced IL-6, osteopontin, monocyte chemotactic protein 1, intercellular adhesion molecule 1, CD68 and tissue factor expression in the arterial wall and lowered plasma IL-6 in diabetic mice. Conclusions: Targeting NFAT signaling may be a novel and attractive approach for the treatment of diabetic macrovascular complications.
eu_rights_str_mv openAccess
format article
id anni_debcb8d7aeeaaa9ec9fb8df2e0f349d6
identifier_str_mv Zetterqvist, A, Berglund, L, Blanco, F, y otros. "Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice". PLoS ONE Vol.8, no.6 [en línea] 2013, doi:10.1371/journal.pone.0065020
instacron_str Universidad de la República
institution Universidad de la República
instname_str Universidad de la República
language eng
language_invalid_str_mv en
network_acronym_str anni
network_name_str oai-lr-anni
oai_identifier_str oai:colibri.udelar.edu.uy:20.500.12008/21978
publishDate 2013
publishDateSort 2013
publisher.none.fl_str_mv PLOS ONE Editorial Board
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rights_invalid_str_mv Licencia Creative Commons Atribución (CC - BY)
spelling Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic MiceZetterqvist, Anna V.Berglund, Lisa M.Blanco, FabianaGarcia-Vaz, ElianaWigren, MariaDuner, PontusDutius Andersson, Anna-MariaNilsson, JanBengtsson, EvaSpegel, PeterObjective of the Study: Diabetic patients have a much more widespread and aggressive form of atherosclerosis and therefore, higher risk for myocardial infarction, peripheral vascular disease and stroke, but the molecular mechanisms leading to accelerated damage are still unclear. Recently, we showed that hyperglycemia activates the transcription factor NFAT in the arterial wall, inducing the expression of the pro-atherosclerotic protein osteopontin. Here we investigate whether NFAT activation may be a link between diabetes and atherogenesis. Methodology and Principal Findings: Streptozotocin (STZ)-induced diabetes in apolipoprotein E2/2 mice resulted in 2.2 fold increased aortic atherosclerosis and enhanced pro-inflammatory burden, as evidenced by elevated blood monocytes, endothelial activation- and inflammatory markers in aorta, and pro-inflammatory cytokines in plasma. In vivo treatment with the NFAT blocker A-285222 for 4 weeks completely inhibited the diabetes-induced aggravation of atherosclerosis, having no effect in non-diabetic mice. STZ-treated mice exhibited hyperglycemia and higher plasma cholesterol and triglycerides, but these were unaffected by A-285222. NFAT-dependent transcriptional activity was examined in aorta, spleen, thymus, brain, heart, liver and kidney, but only augmented in the aorta of diabetic mice. A- 285222 completely blocked this diabetes-driven NFAT activation, but had no impact on the other organs or on splenocyte proliferation or cytokine secretion, ruling out systemic immunosuppression as the mechanism behind reduced atherosclerosis. Instead, NFAT inhibition effectively reduced IL-6, osteopontin, monocyte chemotactic protein 1, intercellular adhesion molecule 1, CD68 and tissue factor expression in the arterial wall and lowered plasma IL-6 in diabetic mice. Conclusions: Targeting NFAT signaling may be a novel and attractive approach for the treatment of diabetic macrovascular complications.PLOS ONE Editorial BoardZetterqvist Anna V.Berglund Lisa M.Blanco FabianaGarcia-Vaz ElianaWigren MariaDuner PontusDutius Andersson Anna-MariaNilsson JanBengtsson EvaSpegel Peter2019-09-27T17:50:07Z2019-09-27T17:50:07Z2013Artículoinfo:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfZetterqvist, A, Berglund, L, Blanco, F, y otros. "Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice". PLoS ONE Vol.8, no.6 [en línea] 2013, doi:10.1371/journal.pone.0065020https://hdl.handle.net/20.500.12008/21978reponame:COLIBRIinstname:Universidad de la Repúblicainstacron:Universidad de la RepúblicaenengPLoS ONE Vol.8, no.6, 2013Las obras depositadas en el Repositorio se rigen por la Ordenanza de los Derechos de la Propiedad Intelectual de la Universidad de la República.(Res. Nº 91 de C.D.C. de 8/III/1994 – D.O. 7/IV/1994) y por la Ordenanza del Repositorio Abierto de la Universidad de la República (Res. Nº 16 de C.D.C. de 07/10/2014)info:eu-repo/semantics/openAccessLicencia Creative Commons Atribución (CC - BY)oai:colibri.udelar.edu.uy:20.500.12008/219782026-04-14T10:28:01Z
spellingShingle Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice
Zetterqvist, Anna V.
status_str publishedVersion
title Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice
title_full Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice
title_fullStr Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice
title_full_unstemmed Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice
title_short Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice
title_sort Inhibition of Nuclear Factor of Activated T-Cells (NFAT) Suppresses Accelerated Atherosclerosis in Diabetic Mice
url https://hdl.handle.net/20.500.12008/21978