Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis

Racemose neurocysticercosis is an aggressive disease caused by the aberrant expansión of the cyst form of Taenia solium within the subarachnoid spaces of the human brain and spinal cord resulting in a mass effect and chronic inflammation. Although expansion is likely caused by the proliferation and...

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主要作者: Orrego, Miguel A (author)
其他作者: Verastegui, Manuela R (author), Vasquez, Carlos M (author), Koziol, Uriel (author), Laclette, Juan P (author), Garcia, Hector H (author), Nash, Theodore E (author)
格式: article
語言:英语
出版: 2021
主題:
在線閱讀:https://hdl.handle.net/20.500.12008/37474
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author Orrego, Miguel A
author2 Verastegui, Manuela R
Vasquez, Carlos M
Koziol, Uriel
Laclette, Juan P
Garcia, Hector H
Nash, Theodore E
author2_role author
author
author
author
author
author
author_browse Garcia, Hector H
Koziol, Uriel
Laclette, Juan P
Nash, Theodore E
Orrego, Miguel A
Vasquez, Carlos M
Verastegui, Manuela R
author_facet Orrego, Miguel A
Verastegui, Manuela R
Vasquez, Carlos M
Koziol, Uriel
Laclette, Juan P
Garcia, Hector H
Nash, Theodore E
author_role author
collection COLIBRI
dc.contributor.none.fl_str_mv Orrego Miguel A
Verastegui Manuela R
Vasquez Carlos M
Koziol Uriel, Universidad de la República (Uruguay). Facultad de Ciencias. Instituto de Biología.
Laclette Juan P
Garcia Hector H
Nash Theodore E
dc.creator.none.fl_str_mv Orrego, Miguel A
Verastegui, Manuela R
Vasquez, Carlos M
Koziol, Uriel
Laclette, Juan P
Garcia, Hector H
Nash, Theodore E
dc.date.none.fl_str_mv 2021
2023-06-07T16:12:54Z
2023-06-07T16:12:54Z
dc.format.none.fl_str_mv 18 h
application/pdf
dc.identifier.none.fl_str_mv Orrego, M, Verastegui, M, Vasquez, C [y otros autores] "Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis". PLOS Neglected Tropical Diseases. [en línea] 2021, 15(3): e0009303. 18 h. DOI: 10.1371/journal.pntd.0009303.
https://hdl.handle.net/20.500.12008/37474
10.1371/journal.pntd.0009303
1935-2735
dc.language.none.fl_str_mv en
eng
dc.publisher.none.fl_str_mv PLOS
dc.relation.none.fl_str_mv PLOS Neglected Tropical Diseases, 2021, 15(3): e0009303.
dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
Licencia Creative Commons Atribución (CC - By 4.0)
dc.source.none.fl_str_mv reponame:COLIBRI
instname:Universidad de la República
instacron:Universidad de la República
dc.subject.none.fl_str_mv Racemose neurocysticercosis
Taenia solium
Serine/threonine-protein kinase plk1
dc.title.none.fl_str_mv Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis
dc.type.none.fl_str_mv Artículo
info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
description Racemose neurocysticercosis is an aggressive disease caused by the aberrant expansión of the cyst form of Taenia solium within the subarachnoid spaces of the human brain and spinal cord resulting in a mass effect and chronic inflammation. Although expansion is likely caused by the proliferation and growth of the parasite bladder wall, there is little direct evidence of the mechanisms that underlie these processes. Since the development and growth of cysts in related cestodes involves totipotential germinative cells, we hypothesized that the expansive growth of the racemose larvae is organized and maintained by germinative cells. Here, we identified proliferative cells expressing the serine/threonine-protein kinase plk1 by in situ hybridization. Proliferative cells were present within the bladder wall of racemose form and absent from the homologous tissue surrounding the vesicular form. Cyst proliferation in the related model species Taenia crassiceps (ORF strain) occurs normally by budding from the cyst bladder wall and proliferative cells were concentrated within the growth buds. Cells isolated from bladder wall of racemose larvae were established in primary cell culture and insulin stimulated their proliferation in a dose-dependent manner. These findings indicate that the growth of racemose larvae is likely due to abnormal cell proliferation. The different distribution of proliferative cells in the racemose larvae and their sensitivity to insulin may reflect significant changes at the cellular and molecular levels involved in their tumor-like growth. Parasite cell cultures offer a powerful tool to characterize the nature and formation of the racemose form, understand the developmental biology of T. solium, and to identify new effective drugs for treatment.
eu_rights_str_mv openAccess
format article
id anni_01ac81c2933b29e180e34d5beb56a4c1
identifier_str_mv Orrego, M, Verastegui, M, Vasquez, C [y otros autores] "Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis". PLOS Neglected Tropical Diseases. [en línea] 2021, 15(3): e0009303. 18 h. DOI: 10.1371/journal.pntd.0009303.
10.1371/journal.pntd.0009303
1935-2735
instacron_str Universidad de la República
institution Universidad de la República
instname_str Universidad de la República
language eng
language_invalid_str_mv en
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oai_identifier_str oai:colibri.udelar.edu.uy:20.500.12008/37474
publishDate 2021
publishDateSort 2021
publisher.none.fl_str_mv PLOS
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rights_invalid_str_mv Licencia Creative Commons Atribución (CC - By 4.0)
spelling Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosisOrrego, Miguel AVerastegui, Manuela RVasquez, Carlos MKoziol, UrielLaclette, Juan PGarcia, Hector HNash, Theodore ERacemose neurocysticercosisTaenia soliumSerine/threonine-protein kinase plk1Racemose neurocysticercosis is an aggressive disease caused by the aberrant expansión of the cyst form of Taenia solium within the subarachnoid spaces of the human brain and spinal cord resulting in a mass effect and chronic inflammation. Although expansion is likely caused by the proliferation and growth of the parasite bladder wall, there is little direct evidence of the mechanisms that underlie these processes. Since the development and growth of cysts in related cestodes involves totipotential germinative cells, we hypothesized that the expansive growth of the racemose larvae is organized and maintained by germinative cells. Here, we identified proliferative cells expressing the serine/threonine-protein kinase plk1 by in situ hybridization. Proliferative cells were present within the bladder wall of racemose form and absent from the homologous tissue surrounding the vesicular form. Cyst proliferation in the related model species Taenia crassiceps (ORF strain) occurs normally by budding from the cyst bladder wall and proliferative cells were concentrated within the growth buds. Cells isolated from bladder wall of racemose larvae were established in primary cell culture and insulin stimulated their proliferation in a dose-dependent manner. These findings indicate that the growth of racemose larvae is likely due to abnormal cell proliferation. The different distribution of proliferative cells in the racemose larvae and their sensitivity to insulin may reflect significant changes at the cellular and molecular levels involved in their tumor-like growth. Parasite cell cultures offer a powerful tool to characterize the nature and formation of the racemose form, understand the developmental biology of T. solium, and to identify new effective drugs for treatment.PLOSOrrego Miguel AVerastegui Manuela RVasquez Carlos MKoziol Uriel, Universidad de la República (Uruguay). Facultad de Ciencias. Instituto de Biología.Laclette Juan PGarcia Hector HNash Theodore E2023-06-07T16:12:54Z2023-06-07T16:12:54Z2021Artículoinfo:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersion18 happlication/pdfOrrego, M, Verastegui, M, Vasquez, C [y otros autores] "Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis". PLOS Neglected Tropical Diseases. [en línea] 2021, 15(3): e0009303. 18 h. DOI: 10.1371/journal.pntd.0009303.https://hdl.handle.net/20.500.12008/3747410.1371/journal.pntd.00093031935-2735reponame:COLIBRIinstname:Universidad de la Repúblicainstacron:Universidad de la RepúblicaenengPLOS Neglected Tropical Diseases, 2021, 15(3): e0009303.Las obras depositadas en el Repositorio se rigen por la Ordenanza de los Derechos de la Propiedad Intelectual de la Universidad de la República.(Res. Nº 91 de C.D.C. de 8/III/1994 – D.O. 7/IV/1994) y por la Ordenanza del Repositorio Abierto de la Universidad de la República (Res. Nº 16 de C.D.C. de 07/10/2014)info:eu-repo/semantics/openAccessLicencia Creative Commons Atribución (CC - By 4.0)oai:colibri.udelar.edu.uy:20.500.12008/374742026-04-14T10:10:06Z
spellingShingle Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis
Orrego, Miguel A
Racemose neurocysticercosis
Taenia solium
Serine/threonine-protein kinase plk1
status_str publishedVersion
title Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis
title_full Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis
title_fullStr Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis
title_full_unstemmed Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis
title_short Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis
title_sort Identification and culture of proliferative cells in abnormal Taenia solium larvae: Role in the development of racemose neurocysticercosis
topic Racemose neurocysticercosis
Taenia solium
Serine/threonine-protein kinase plk1
url https://hdl.handle.net/20.500.12008/37474